Microbiology Bacteria Paper

I would not rate science is storybook fun, but who knew it become a mystery. Trying to find forth what was in our number seven vial would become a appointment we were willing to take on. As I began the test of deciding if our humble bacteria jock was gram positive or negative, Jordan my science teammate, was putting together a smear plate. In as footling as ten minutes we had discovered by the pink oval shapes we were observing, our seed friend was a gram negative rod. We had narrowed our search chain reactor to five We decided next we would do oxygen along with a motility test. Along with those, we did a fermentation investigation.These tests would narrow our pursuit garbage down even further. Unfortunately these tests take time. The following day, we curiously went to our stash of experiments. As we observed we soon realized our little bacteria was a non-motile, facultative anaerobe. With the observation of acid and gas formation, this microorganism was able to catabolize glucose, milk sugar and fructose. Our smear plate, with white convex muciod look colonies confirmed our suspicion. We were nowadays looking at vial number seven with anew respect, he now had a name, and that name was Klebsiella pneumonia.Read as well as Lab 2 biologyNow that our mystery bug had a proper name, where would we find it? This bacterium, I found out is ubiquitous in nature, meaning it seems to be present, e verywhere at the same time. Just about anywhere you would step, touch or see in nature you could bet this little guy is there. Its family is verdant in soil, water and vegetables. But they have cousins, uncles, second cousins etc. honorable about everywhere else. Wow, was all I could muster. But our friend not only if had a large family in nature, this microbe also found residence inside the gentleman body.The respiratory, intestinal, and urogenital tracts atomic number 18 a favored neighborhood for this microscopic organism. This tiny microbe seems to find unspoiled about any region environmentally friendly. Not only is it very versatile in were it lives, I would soon learn it was also rightful(prenominal) as versatile in what is able to do. This small bacterium could ruin the human body if allowed to. Our secret microbe was considered to be an opportunistic human pathogen, meaning that under certain conditions it may cause disease. All this little guy needed was the perfect scenario and he could thrive in only a way harmful bacterium could.Read Chapter 8 microbic GeneticsPersons with underlying diseases such as alcoholism or lung infections were more or less of their favorite captives. Along with people who argon hospitalized and receive invasive procedures, world their other prime hostages. These guys are on the top ten close to known list of nosocomial infection pathogens. Trying to get relieve of this pathogen is no easy feat either Once this type is allow in he really does not want to leave. Of course, the first storm t o rid the body of this pathogen is to use a timely and aggressive handling of antibiotics.Even that though, can prove to not be beneficial. Since these little critters are proven to be resistant, meaning, they develop ways to inactivate or annul the antibiotic. Many cases have to be treated with cephalosporins and aminoglycosides to slide by the bacteria a two-pronged attack. This bacterium certainly has a powerful military when dispersed. But how does this little fella do this? Well, the pathology for it to become Pneumonia develops when the bacilli invade and cover within the alveolar consonant spaces.General Biology Ii Study Guide (Online Class)The pulmonary parenchyma becomes consolidated, and the mucoid exudates that fill the alveoli is controlled by macrophages, fibrin, and edema fluid. Neutrophils, our bodies own fighting soldiers are inhibited by a neutral polysaccharide in the contraction of this bacterium. Numerous encapsulated gram-negative bacilli appear free in th e exudates and in alveolar macrophages. Then exudates accumulate and the alveolar wall becomes compacted and healthy tissue starts to degrade. This area, where the microbe does its best work, is the area of gas exchange with the blood, a vital diverge of the human anatomy/physiology.So when this microbe is able to take over, a hefty defense force of antibacterial agents have to come to the battle. Antibiotics themselves though have a become an overused tool. This has become a growing task and many actions have now come into play to stop the maltreatment of these products. Some would argue a little too late. This lapse of abject judgment has led to the numerous strains of antibiotic resistant infections. Klebsiella pneumonia is one of the pencil lead culprits. The thing is though, bacteria does not know its playing a spartan game with us, all bacteria is trying to do is win.References Klebsiella pneumonia . Retrieved from http//www.klebsiellapneumoniae.org/ (Klebsiella pneumoni ae , 2011) Klebasiella pneumonia. Retrieved from http//www.histopathology-india.net/Klebsiella_pneumoniae.htm (Dr. Sampuna Roy, 2011)